HEXIM1
- Aliases
-
- CLP-1
- CLP1
- Cardiac lineage protein 1
- EDG1
- Estrogen down-regulated gene 1 protein
- HEXIM1
- HIS1
- HMBA-inducible
- Hexamethylene bis-acetamide-inducible protein 1
- MAQ1
- Menage a quatre protein 1
- cardiac lineage protein 1
- estrogen down-regulated gene 1 protein
- hexamethylene bis-acetamide inducible 1
- hexamethylene bis-acetamide-inducible protein 1
- hexamethylene bisacetamide-inducible protein
- hexamethylene-bis-acetamide-inducible transcript 1
- hexamthylene bis-acetamide inducible 1
- menage a quatre 1
- menage a quatre protein 1
- protein HEXIM1
- Description
- HEXIM1 is an intronless transcriptional regulator which functions as a general RNA polymerase II transcription inhibitor. HEXIM1 expression is induced by hexamethylene-bis-acetamide in vascular smooth muscle cells.
Attributes
- QA State
- Curated
- Type
- Protein
- HGNC Name
- HEXIM1
- Certifications
-
- None
- QA State for Breast
- Under Review
Non-Public Biomarker
Organ-specific information for this biomarker is currently being annotated or is "under review". Logging in may give you privileges to view additional information. Contact the Informatics Center if you believe you should have access.
Non-Public Biomarker
Organ-specific information for this biomarker is currently being annotated or is "under review". Logging in may give you privileges to view additional information. Contact the Informatics Center if you believe you should have access.
- Development and validation of sandwich ELISA microarrays with minimal assay interference.
- Discovery and preliminary confirmation of novel early detection biomarkers for triple-negative breast cancer using preclinical plasma samples from the Women's Health Initiative observational study.
- Plasma biomarker profiles differ depending on breast cancer subtype but RANTES is consistently increased.
Non-Public Biomarker
Organ-specific information for this biomarker is currently being annotated or is "under review". Logging in may give you privileges to view additional information. Contact the Informatics Center if you believe you should have access.