PCAT1

Aliases
  • HSALNG0068395
  • HSALNG0068398
  • HSALNG0068414
  • HSALNG0068435
  • NONHSAG051242
  • NONHSAG051246
  • NONHSAG051248
  • NONHSAG051250
  • PCA1
  • PCAT-1
  • PCAT1
  • Prostate Cancer Associated Transcript 1
  • Prostate Cancer Associated Transcript 1 (Non-Protein Coding)
Description
From NCBI Gene: This gene produces a long non-coding RNA that promotes cell proliferation and is upregulated in prostate, colorectal, and other cancers. This RNA negatively regulates the BRCA2 tumor suppressor protein and positively regulates Myc oncoprotein. It contains binding sites for microRNAs, and may act as a sponge for microRNAs that regulate cell growth pathways. [provided by RefSeq, Dec 2017]
Attributes
QA State
Under Review
Type
Epigenetic
HGNC Name
PCAT1
Certifications
  • None
QA State for Prostate
Under Review

 Non-Public Biomarker

Organ-specific information for this biomarker is currently being annotated or is "under review". Logging in may give you privileges to view additional information. Contact the Informatics Center if you believe you should have access.

 Non-Public Biomarker

Organ-specific information for this biomarker is currently being annotated or is "under review". Logging in may give you privileges to view additional information. Contact the Informatics Center if you believe you should have access.

 Non-Public Biomarker

Organ-specific information for this biomarker is currently being annotated or is "under review". Logging in may give you privileges to view additional information. Contact the Informatics Center if you believe you should have access.

 Non-Public Biomarker

Organ-specific information for this biomarker is currently being annotated or is "under review". Logging in may give you privileges to view additional information. Contact the Informatics Center if you believe you should have access.

 Non-Public Biomarker

Organ-specific information for this biomarker is currently being annotated or is "under review". Logging in may give you privileges to view additional information. Contact the Informatics Center if you believe you should have access.